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Stanford AIMI 63

Interval placement of an additional coronary sinus lead for a biventricular AICD.

Known rib fractures, previously identified.

Bilateral basal atelectasis without additional significant pulmonary findings.

Mild prominence of upper zone vessels, possibly related to low-inspiratory volumes.

No change in the position of lines and wires, with a segment of the lead overlying the left upper lung region.

Increasing basilar pleural fluid, more pronounced on the left side.

Bilateral patchy airspace opacities, which may represent infection or pulmonary edema.

New insertion of a feeding tube.

Persistent opacities in the left greater than right bibasilar regions.

Reticular prominence suggesting either chronic interstitial lung disease or mild pulmonary edema.

Slight improvement in aeration in the left mid lung, with persistent moderate pulmonary edema.

Low lung volumes with right hemidiaphragm elevation and bibasilar opacities, likely representing atelectasis, though infection cannot be excluded.

Mild pulmonary vascular congestion, appears stable.

Persistent bibasilar opacities, which may represent compressive atelectasis or consolidation.

Remaining skeletal structures appear grossly normal.

Increased interstitial prominence suggestive of mild interstitial edema.

No acute pulmonary fractures identified.

Stable position of the right internal jugular (IJ) line, nasogastric tube, and left upper extremity peripherally inserted central catheter (PICC line).

Increasing right basilar opacification, possibly indicative of developing pneumonia or atelectasis.

Presence of endotracheal tube approximately 4.5 cm above the carina, nasogastric tube, right-sided internal jugular catheter, Swan-Ganz catheter via right internal jugular approach, mediastinal drains, and intact sternal wires, indicating postoperative status.

New endotracheal tube located approximately 3 cm above the carina.

Surgical clips are present in the right upper lobe.

Relatively low lung volumes without associated pathology

Bilateral costophrenic sulci remain blunted, which may represent small pleural effusions or pleural thickening.

Right hilar mass suggestive of a significant pathology.

Status post removal of right-sided pigtail catheter.

Chest tube and right jugular line previously noted have been removed.

Unchanged position of the right upper extremity peripherally inserted central catheter (PICC) with its tip at the atrio-caval junction.

Small left-sided pneumothorax with chest tube in place.

Degenerative changes and deformity involving the shoulder joints, partially imaged.

Large mass in the right hemithorax mostly unchanged.

Examination otherwise unchanged.

Unchanged appearance of the postoperative left hemithorax.

Unchanged severe cardiomegaly presumed.

Known compression deformity of the T2 vertebral body is not well visualized on the current radiographs.

No evidence of increased pleural fluid.

Left paratracheal opacity, unclear if related to the pulmonary malignancy.

Right peripherally inserted central catheter (PICC) line unchanged, terminating at the superior cavoatrial junction.

Unremarkable, except for surgical clips in the right upper quadrant suggestive of prior cholecystectomy.

Increased right infrahilar interstitial pattern, likely indicative of chronic pulmonary changes.

Interval placement of the stent graft from the proximal to mid descending thoracic aorta with resulting decreased prominence of the superior mediastinum.

Bandlike consolidation extending from a masslike structure at the right hilum to the adjacent pleura.

Unchanged retrocardiac opacification with left-sided pleural effusion.

Vague right lower lung peripheral opacity.

Increased right basilar opacity and pleural effusion, suggestive of an early consolidative process or atelectasis.

No significant change in the bilateral pleural effusion when compared to the previous study.

Alveolar opacity in the middle right lung field, suggesting an infectious or inflammatory process.

Small regions of linear atelectasis at the base of the left lung.

Mild cardiomegaly with findings consistent with known mediastinal lymphadenopathy.

Presence of surgical clips along the lower mediastinum and over the lateral right chest.

No evidence of focal consolidation or pleural effusions in the chest. Cardiomediastinal silhouette is unremarkable. Presence of multiple surgical clips in the left upper quadrant, possibly related to prior splenectomy. An intramedullary rod is noted within the right humerus.

Prominent thoracic aorta and widened mediastinum, which may suggest mediastinal pathology.

Nodular opacity projecting over the right lower lobe

Hyperinflated lungs with flattening of the hemidiaphragms suggestive of COPD.

Nodule in the right lower lobe.

No acute pulmonary findings. Retrocardiac region not well assessed due to overlying soft tissues/technique.

Streaky opacity across the right lower lung consistent with right middle lobe atelectasis

Endotracheal tube is appropriately positioned at the level of the mid-clavicular heads.

Normal chest radiograph without evidence of tuberculosis.

Old posterior right rib fractures identified.

Cardiac silhouette enlargement, which may be exaggerated by the imaging technique.

The left tibia and fibula appear normal.

Pulmonary congestion and pleural effusion noted, left greater than right, unchanged from prior imaging.

Unchanged moderate right pleural effusion, partially loculated.

Right-sided central catheter terminating in the right atrium.

Stable small-to-moderate right pleural effusion with no change in volume following the removal of pigtail pleural drain.

Supportive equipment and postsurgical changes with no interval change.

Endotracheal tube tip is 6 cm from the carina, which is acceptable given the neutral or elevated position of the chin.

Unusual appearance of the bones with sclerosis and altered corticomedullary differentiation, which may suggest an underlying metabolic process such as renal osteodystrophy, myelofibrosis, or sickle cell disease. Clinical correlation is recommended.

Significantly worsened degenerative changes of the thoracic spine with large anterior flowing osteophytes

Marked elevation of the right hemidiaphragm with small bilateral pleural effusions.

Study of limited evaluative value due to overpenetration.

Redemonstrated osteopenia with multilevel vertebral body compression deformities, unchanged from prior examination.

Multiple old left-sided rib deformities and evidence of previous thoracotomy.

Slight increase in lucency in the left upper and mid hemithorax, suggestive of emphysema.

Improvement in bibasilar reticular interstitial markings, possibly reflecting a response to treatment for nonspecific interstitial pneumonia (NSIP).

Persistent low lung volumes with abnormal opacities in bilateral lung bases and a small left-sided pleural effusion.

Presence of a left internal jugular central catheter with the tip located in the right atrium.

Removal of endotracheal tube, nasogastric tube, and right internal jugular line since the prior study.

Hyperexpansion suggesting underlying chronic obstructive pulmonary disease.

Bibasilar pleural catheters are unchanged in position.

Possible peribronchial opacification in both lower lungs, raising concerns for aspiration or at least atelectasis.

Bilateral pleural effusions, with interval increase.

Stable minimal atelectasis in the left lower lobe.

Suggested mild-to-moderate cardiomegaly.

Compression of multiple thoracic vertebral bodies.

Moderate cardiomegaly, stable to slightly more pronounced.

Multiple left-sided rib fractures without evidence of pneumothorax or left pleural effusion. Mild opacity along the periphery of the lung may suggest contusion. Plate-like atelectasis is present in the left lower lung field. Cardiomediastinal silhouette and hila are unremarkable.

No mediastinal shift or enlargement.

Increased opacity at the right lung base, suggestive of atelectasis or infection.

Patchy right basilar opacity, with no clear evidence of consolidation on the lateral view.

No evidence of viral lung involvement by COVID-19 on the current examination.

Mild opacities in bilateral lower lobes suggestive of atelectasis or pneumonia.

Interval progression of right middle lobe atelectasis

Interval decrease in bibasilar opacities, with residual opacity suggestive of either atelectasis or consolidation.

Left retrocardiac infiltrate and/or atelectasis.

Left lower lobe demonstrates poor aeration, likely due to relaxation atelectasis secondary to the effusion.

Prominent bibasilar atelectasis with blunting of the left costophrenic angle, which may represent a small pleural effusion or pleural thickening. No significant effusion is identified on the lateral view.

Evidence of several old, healed rib fractures on the right.

Mild interstitial abnormality in the lower lungs, likely due to mild cardiac decompensation.